The Cause of the Rapid Increase of Type 2 Diabetes

Type 2 diabetes is among the most hastily increasing chronic ailments worldwide. The call for its prime prevention has been progressively been emphasized, though merely during the past 10 years. The key reasons to prevent Type 2 diabetes are the potential deterrence or delay of complications connected to Type 2 diabetes to lessen both the socio-economic burden and human suffering in the society. It has been frequently shown that both asymptomatic and symptomatic diabetic patients have an augmented occurrence of both microvascular and macrovascular impediment by the time the ailment is first diagnosed.

The growth of diabetes in the US has arrived at epidemic scale. According to the report from Centers for Disease Control and Prevention, more than half of adults are suffering from diabetes and around 17 percent of teens (ages 2-19) and children have obesity. The studies have revealed that chronic diseases, such as diabetes and heart disease, are very common in the U.S. in comparison to European nations. This is partially, since more Americans practice unhealthy behaviours than Europeans do. The World Health Organization revealed that chronic disease is accountable for around 60 percent of all deaths recorded worldwide (American Diabetes Association 91).

The amplified knowledge regarding the pathogenesis, etiology, and natural account of type 2 diabetes has contributed to key prevention becoming an authenticity. Though an unambiguously conventional consensus about the untimely pathogenesis is lacking, protective measures could be based upon the finest present available acquaintance. The quickly rising number of patients suffering from Type 2 diabetes, the harshness of the illness, its severe, multiple complications, and the growing socio-economic outlay emphasize the value of instant preventive measures. The present state for type 2 diabetes could be compared with the plague of coronary heart ailment throughout the 1960s and 1970s in numerous industrialized countries.

Diabetes is highly associated with certain types of food, particularly, junk food. Junk food can have significant impact on someone’s health. However, many people enjoy junk food because of its great taste. Health consequences of such food can be serious and costly in the end period. Health effects can range from the mere rapid weight gain that is not painful to some other terminal illnesses. Taking junk food is another way of attracting medical attention through therapies to reduce high blood pressure, stabilize sugar levels and weight loss among many others. Visiting medical officers and healthcare experts can be disastrous (Shafer Sherri, 2001).

Junk foods are known for damaging the pancreas. A damage to the pancreas means that a consumer of junk food will be exposed to a range of disorders including diabetes mellitus. Insulin is affected easily by the components that are present in these hamburgers and spam. Studies have noted that junk food have high levels of oil and fat. Too much fat and oil in foods hampers complete digestion of foods in the stomach. Digestive juices do not work well when such fats are in high concentration in foods. For adequate digestion then high amounts of bloods and enzymes will be required in the body.

The Government role of the United States plays a major function in the manufacture, safeguard, and delivery of food. The government regulates the appropriate preparation and storage of food. The implementation of these policies has been greatly affected by the civic protest following an outbreak of food poisoning. Consequently, the examination of foods has turned into a responsibility of the US government.

The rate of type 2 diabetes rises with increasing age, and the immense majority of diabetic issues is the aged. There are physiological grounds for this and they are greatly connected to lifestyle. Muscle mass however, typically decreases with aging. To what degree this can be seen as a part of a usual ageing process, and to what degree it is due to an augment in inactive lifestyle with age is not completely settled. In addition, a variety of musculoskeletal problems augments with age and repeatedly affect exercise behaviour.

It is likely that a number of exposures throughout the intra-uterine phase and early childhood may stop normal β-cell replication and growth leading to a permanently abridged β-cell ability. The pancreatic β-cell decreases with age. Peripheral insulin resistance due to influence such as physical inactivity, diet, and obesity accelerate this process. Most people who are genetically vulnerable to diabetes, have the process that leads to hyperglycemia and, eventually, to medical diabetes. The pace of this growth and hence the age-at-onset of illness are clearly determined by the strength of ecological exposure.

There are several target groups that type 2 diabetes seems to develop prevalent cases in the society. Family history affects the rise of this disease and disorder among the people in the environment. Medical officers note that the accepted pathogenic model is only based upon some assumption about genetic predisposition but rather not a real cause for this disease. Those individuals carrying diabetes susceptibility genes tend to develop type 2 diabetes faster compared to others in the society. For instance, Pacific islanders and some Native Americans have a lifetime prevalence rate of over 50% of type 2 diabetes. These populations are assumed to possess high levels of thrifty genotypes that other populations do not have across the world. Other studies show that people aged 70 years and above show high cases of type 2 diabetes. However, other studies suggest that this type of diabetes may not affect all the family members within a given social setting (World Health Organization).

Many studies associate high cases of type 2 diabetes with early childhood malnutrition. People may improve their nutritional levels thinking that it reduces case of this disease. However, those with the effects of diabetes-susceptible genes may not improve their conditions even with good nutrition. Protein metabolism among pregnant mothers is the major cause of this disease. Amino acids during pregnancy should be monitored because their levels may affect the fetus. Due to this, adequate fetal programming must be done to prevent further development of this disease. Low birth weights largely contribute to high cases of type 2 diabetes among children. It is vital to control pregnancy or fetal growth through hormones, placental functions and other factors just keep the weight of the unborn in the right position and on a clear development path.

Childhood obesity is one factor that exacerbates the rising cases of type 2 diabetes among the youth and the adults. Health officers and parents should identify those with high risks of obesity in their later lives. This will trigger adequate prevention mentions before it becomes too late for them in the society. Population approaches must aim to avoid obesity among human beings. Health professionals and other medical institution should provide a clear guideline on how to keep obesity levels to controllable levels. This will improve the overall health of the community because dealing with children is often cheaper, if compared to other groups in the society.

After pregnancy, women risk developing type 2 diabetes because of hyperglycemia or gestational diabetes during pregnancy. Incidences per year are around 5% among pregnant mothers. However, gestational diabetes does not affect the mother alone but still the offspring risk contracting type 2 diabetes. Pregnant women require adequate management of their foods and exercises so that they improve their physical activities. This will promote circulation of the blood and help keep risk levels of gestational diabetes to zero. However, those women with previous cases of gestational diabetes should be placed under systematic monitoring and control to keep glucose levels down (Kohei Kaku ).

Medical studies show that people with any level of hypertension tend to develop diabetes immediately. The risk of cardiovascular diseases tends to go up among hypertensive patients. It is however, necessary to determine glucose levels at regular intervals. Control of blood pressure alone is not adequate in controlling development of type 2 diabetes among hypertension patients. Adult obesity and physical inactivity are some of the most modifiable risk factors that lead to type 2 diabetes in the society. The control of adult obesity can reduce type 2 diabetes by about 50% on average. In addition, control of physical inactivity among adults can reduce risks of this disease by around 30%. However, the primary focus should be on weight management of adults. Physical activity should be stimulated so that adults improve their metabolic rates.

There are a number of stages for the development of this type 2 diabetes. The early stages are abnormalities caused by glucose homeostasis. People with slightly elevated blood glucose levels develop frank diabetes. Treatment and prevention of glucose-related problems will assist in reducing high cases of type 2 diabetes in the environment. The history of many prevention programs matters to reducing the cases of type 2 diabetes. Other measures of controlling other health conditions such as obesity and weight loss determine the prevalence rates of type 2 diabetes among human beings. Adequate education should be provided to inform people about risk factors for this type of disease.

Issues to do with proper diet require close attention because in the end they will reduce risk factors of developing type 2 diabetes. Many risk factors of type 2 diabetes lately come from poor primary prevention measures. Type 2 diabetes needs serious attention in the present, as there were few cases across the world the past. Medical experts offer secondary prevention strategies that do not provide full treatment. Primary prevention measures contribute to full recovery of patients with type 2 diabetes. Prevention measures, based on population approach, are necessary in reducing the risks of type 2 diabetes (NP Steyn et al).

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